Search results for " Energy metabolism"

showing 10 items of 14 documents

Physical activity and fat-free mass during growth and in later life.

2021

ABSTRACT Background Physical activity may be a way to increase and maintain fat-free mass (FFM) in later life, similar to the prevention of fractures by increasing peak bone mass. Objectives A study is presented of the association between FFM and physical activity in relation to age. Methods In a cross-sectional study, FFM was analyzed in relation to physical activity in a large participant group as compiled in the International Atomic Energy Agency Doubly Labeled Water database. The database included 2000 participants, age 3–96 y, with measurements of total energy expenditure (TEE) and resting energy expenditure (REE) to allow calculation of physical activity level (PAL = TEE/REE), and cal…

Adipose Tissue/metabolismMaleAgingIMPACTMedicine (miscellaneous)Medical and Health SciencesLONGITUDINAL CHANGESRC12000302 clinical medicineEngineeringenergy expenditureFaculty of Science80 and overMedicineWATER030212 general & internal medicineChildInternational Atomic Energy Agency Doubly Labeled Water database groupAged 80 and overNutrition and DieteticsMiddle Aged3142 Public health care science environmental and occupational healthEditorialAdipose TissueChild PreschoolFemalephysical activity levelPeak bone massAdultAdolescentPhysical activityBONE MASS030209 endocrinology & metabolismDoubly labeled waterFat massVDP::Medisinske Fag: 700::Helsefag: 800::Ernæring: 81103 medical and health sciencesYoung AdultAnimal scienceTotal energy expenditureFat free massClinical ResearchBENEFITSHumansResting energy expenditure/dk/atira/pure/core/keywords/TheFacultyOfSciencePreschoolExerciseNutritionAgedbody compositionNutrition & DieteticsAdipose Tissue/metabolism; Adolescent; Adult; Aged; Aged 80 and over; Body Composition; Child; Child Preschool; Cross-Sectional Studies; Energy Metabolism; Exercise; Female; Humans; Male; Middle Aged; Young Adult; age; body composition; doubly labeled water; energy expenditure; physical activity levelbusiness.industryPreventionPhysical activity leveldoubly labeled water3141 Health care scienceCross-Sectional StudiesageLEAN BODY-MASSYOUNGWEIGHTbusinessEnergy Metabolismhuman activities
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Influence of family history of NIDDM on basal metabolic rate in sedentary and active women

2011

Aim and methods: The influence of family history of non-insulin-dependent diabetes (NIDDM) on basal metabolic rate (BMR) has been investigated in 116 voluntarily women: 25 sedentary and 34 athletes without a family predisposition to type 2 diabetes (FH-); 21 sedentary and 15 athletes with a second degree predisposition to NIDDM (FH +); 10 sedentary and 11 athletes with a first degree predisposition to NIDDM (FH ++). Results: The results showed that family history on type 2 diabetes is strongly related to sedentary in women with significant high body weight values and an increased fat mass. There was no significant difference in the body parameters among the athletes groups, confirming the p…

AdultDiabetes Mellitus Type 2Case-Control StudiesBody CompositionHumansFemaleBasal MetabolismExerciseBody composition Diabetes mellitus Energy metabolism Epidemiology Motor activity Risk factors Type 2
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What are the roles of calorie restriction and diet quality in promoting healthy longevity?

2014

Epidemiological and experimental data indicate that diet plays a central role in the pathogenesis of many age-associated chronic diseases, and in the biology of aging itself. Data from several animal studies suggest that the degree and time of calorie restriction (CR) onset, the timing of food intake as well as diet composition, play major roles in promoting health and longevity, breaking the old dogma that only calorie intake is important in extending healthy lifespan. Data from human studies indicate that long-term CR with adequate intake of nutrients results in several metabolic adaptations that reduce the risk of developing type 2 diabetes, hypertension, cardiovascular disease and cance…

Agingmedicine.medical_specialtyDisease preventionTime Factorsmedia_common.quotation_subjectLongevityCalorie restrictionNutritional StatusPhysiologyType 2 diabetesDiseaseBiologyVegetarian dietBiochemistryCalorie restriction Diet quality Health Lifespan Vegetarian diet Disease preventionLife ExpectancyNutrientNeoplasmsInternal medicinemedicineAnimalsHumansLife StyleMolecular BiologyCalorie restrictionCaloric Restrictionmedia_common2. Zero hungerLifespanAge FactorsLongevityCalorie restriction; Diet quality; Disease prevention; Health; Lifespan; Vegetarian diet; Age Factors; Aging; Animals; Cardiovascular Diseases; Energy Metabolism; Humans; Longevity; Neoplasms; Nutritional Status; Time Factors; Caloric Restriction; Diet; Life Expectancy; Life Style; Risk Reduction BehaviorVegan Dietmedicine.diseaseDiet3. Good healthEndocrinologyNeurologyHealthCardiovascular DiseasesDietary Reference IntakeAnimal studiesEnergy MetabolismRisk Reduction BehaviorDiet qualityBiotechnology
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Evidence for hypothalamic ketone bodies sensing: impact on food intake and peripheral metabolic responses in mice

2016

Monocarboxylates have been implicated in the control of energy homeostasis. Among them, the putative role of ketone bodies produced notably during high-fat diet (HFD) has not been thoroughly explored. In this study, we aimed to determine the impact of a specific rise in cerebral ketone bodies on food intake and energy homeostasis regulation. A carotid infusion of ketone bodies was performed on mice to stimulate sensitive brain areas for 6 or 12 h. At each time point, food intake and different markers of energy homeostasis were analyzed to reveal the consequences of cerebral increase in ketone body level detection. First, an increase in food intake appeared over a 12-h period of brain keton…

Blood GlucoseMale0301 basic medicineobesitynervous-systemPhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismKetone BodiesEnergy homeostasisEatingMicebodiesHomeostasisGlucose homeostasisoxidative stressAgouti-Related ProteinNeuropeptide YPhosphorylationmonocarboxylate transporters2. Zero hunger[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]fat massHypothalamusKetone bodiesStarvation responseketogenic mediterranean dietweight-lossmedicine.medical_specialtybeta-hydroxybutyrateHypothalamusBiologyDiet High-Fat03 medical and health sciencesInsulin resistancerat-brainPhysiology (medical)Internal medicinemedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Animalsglucose homeostasisAdenylate Kinase/metabolism; Agouti-Related Protein/metabolism; Animals; Blood Glucose; Diet High-Fat; Eating/drug effects; Eating/physiology; Energy Metabolism/drug effects; Energy Metabolism/physiology; Gluconeogenesis/drug effects; Gluconeogenesis/physiology; Homeostasis; Hypothalamus/drug effects; Hypothalamus/metabolism; Insulin Resistance/physiology; Ketone Bodies/pharmacology; Male; Mice; Mice Inbred C57BL; Neuropeptide Y/metabolism; Phosphorylation/drug effectsenergy homeostasisAdenylate KinaseGluconeogenesismedicine.diseaseMice Inbred C57BL030104 developmental biologyEndocrinologyGluconeogenesislow-carbohydrateInsulin ResistanceEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionHomeostasis
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Influence of substrate oxidation on the reward system, no role of dietary fibre.

2011

International audience; It has been suggested that a high intake of dietary fibre helps regulate energy intake and satiety. The present study aimed to examine whether dietary fibre influenced the liking and wanting components of the food reward system, the metabolic state or subsequent intake. Five sessions involving 32 normal-weight subjects (16 men and 16 women, 30.6 ± 7.6 year) were held. The sessions differed in the composition of the bread eaten during breakfasts (dietary fibre content varied from 2.4 to 12.8 g/100 g). Several factors such as the palatability, weight, volume, energy content and macronutrient composition of the breakfasts were adjusted. Energy expenditure, the respirato…

Dietary FiberMaleMESH: Oxidation-ReductionMESH: Dietary Carbohydrates030309 nutrition & dietetics[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionsatietyAppetiteMESH: Food HabitsMESH: Energy IntakeChoice BehaviorMESH: EatingEatingIngestionMESH : FemaleFood sciencePalatabilityMESH : Body WeightGeneral PsychologyMESH : Food Habitsmedia_commonMorning2. Zero hunger0303 health sciencesMESH : Food PreferencesNutrition and Dieteticsdietary fibre05 social sciencesdigestive oral and skin physiologyMESH: Energy MetabolismMESH : Feeding BehaviorBreadMESH : AdultMESH : Dietary Carbohydratesreward systemMESH: Young AdultMESH: Feeding BehaviorFemalePsychologyOxidation-Reductionfood preferencesMESH : AppetiteAdultMESH : EatingMESH : Malemedia_common.quotation_subjectMESH : Young AdultSatiationMESH: Choice BehaviorMESH: BreadYoung Adult03 medical and health sciencesReward systemsensation de faimMESH : Choice BehaviorDietary CarbohydratesHumans0501 psychology and cognitive sciences050102 behavioral science & comparative psychologyMESH: Food PreferencesMESH : Oxidation-ReductionMESH: HumansMESH: SatiationMESH : HumansBody WeightDietary fibreMESH : Energy IntakeAppetiteMESH: AdultFeeding BehaviorMESH: MaleMESH: Body WeightMESH : Energy MetabolismRespiratory quotientMESH: Dietary FiberEnergy densityMESH: AppetiteMESH : Dietary FiberEnergy IntakeEnergy MetabolismMESH : BreadmetabolismMESH: Female[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Satiation
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The neural feedback loop between the brain and adipose tissues

2009

Communication également publiée dans le livre "Adipose tissue development: from animal models to clinical conditions" (ISBN 978-3-8055-9450-9) de C. Levy-Marchal et L. Pénicaud (eds); There are more and more data supporting the importance of nervous regulation of both white and brown adipose tissue mass. This short paper will review the different physiological parameters which are regulated such as metabolism (lipolysis and thermogeneis), secretory activity (leptin and other adipokines) but also to plasticity of adipose tissues (proliferation differentiation and apoptosis). The sensory innervation of white adipose issue and its putative role will be also described. Altogether these results …

MESH: Feedback Physiological[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionPhysiologicalAdipokineAdipose tissueWhite adipose tissueBiologyAutonomic Nervous SystemMESH : Adipose TissueEnergy homeostasisMESH : Autonomic Nervous SystemFeedbackMESH: Autonomic Nervous System[ SDV.NEU.SC ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesMESH: BrainBrown adipose tissuemedicineLipolysisAnimalsHumansMESH: AnimalsComputingMilieux_MISCELLANEOUSFeedback PhysiologicalMESH: HumansLeptinMESH : HumansMESH: Energy MetabolismBrain[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesMESH : Feedback PhysiologicalNeurosecretory SystemsCell biologyMESH : Energy MetabolismAutonomic nervous systemmedicine.anatomical_structureMESH : BrainAdipose TissueMESH: Neurosecretory SystemsMESH : AnimalsEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Neurosecretory Systems[SDV.NEU.SC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesMESH: Adipose Tissue
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Apelin treatment increases complete Fatty Acid oxidation, mitochondrial oxidative capacity, and biogenesis in muscle of insulin-resistant mice.

2012

Both acute and chronic apelin treatment have been shown to improve insulin sensitivity in mice. However, the effects of apelin on fatty acid oxidation (FAO) during obesity-related insulin resistance have not yet been addressed. Thus, the aim of the current study was to determine the impact of chronic treatment on lipid use, especially in skeletal muscles. High-fat diet (HFD)-induced obese and insulin-resistant mice treated by an apelin injection (0.1 μmol/kg/day i.p.) during 4 weeks had decreased fat mass, glycemia, and plasma levels of triglycerides and were protected from hyperinsulinemia compared with HFD PBS-treated mice. Indirect calorimetry experiments showed that apelin-treated mice…

MESH: Oxidation-Reduction[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismGlucose uptakeAMP-Activated Protein KinasesInbred C57BLMice0302 clinical medicineAMP-activated protein kinaseMESH : Lipid MetabolismHyperinsulinemiaMESH: AnimalsMESH: AMP-Activated Protein KinasesMESH : Muscle SkeletalMESH : Fatty AcidsBeta oxidationMESH: Lipid Metabolism0303 health sciencesMESH: Muscle SkeletalbiologyMESH : Diet High-FatFatty AcidsMESH: Energy MetabolismMESH : AMP-Activated Protein KinasesMESH: Mitochondria MuscleSkeletal3. Good healthApelinMitochondriaMESH: Fatty AcidsMESH : Cyclic AMP-Dependent Protein KinasesMESH: Insulin ResistanceAlimentation et NutritionApelinIntercellular Signaling Peptides and ProteinsMuscleMESH : Insulin ResistanceOxidation-Reductionmedicine.medical_specialtyMESH : Mitochondria Muscle030209 endocrinology & metabolismMESH : Mice Inbred C57BLMESH: Cyclic AMP-Dependent Protein KinasesDiet High-Fat03 medical and health sciencesInsulin resistanceAdipokinesMESH: Mice Inbred C57BLInternal medicineMESH : MiceInternal MedicinemedicineFood and NutritionAnimalsMuscle SkeletalMESH: Intercellular Signaling Peptides and ProteinsMESH: MiceMESH : Intercellular Signaling Peptides and Proteins030304 developmental biologyMESH : Oxidation-ReductionAMPKmedicine.diseaseLipid MetabolismCyclic AMP-Dependent Protein KinasesMitochondria MuscleDietMice Inbred C57BLMESH : Energy Metabolism[SDV.AEN] Life Sciences [q-bio]/Food and NutritionAMP-Activated Protein Kinases;Animals;Cyclic AMP-Dependent Protein Kinases;Diet;High-Fat;Energy Metabolism;Fatty Acids;Insulin Resistance;Intercellular Signaling Peptides and Proteins;Lipid Metabolism;Mice;Inbred C57BL;Mitochondria;Muscle;Skeletal;Oxidation-ReductionHigh-FatMESH: Diet High-FatMetabolismEndocrinologyMitochondrial biogenesisbiology.proteinMESH : AnimalsInsulin ResistanceEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Leigh syndrome due to compound heterozygosity of dihydrolipoamide dehydrogenase gene mutations. Description of the first E3 splice site mutation.

2003

Item does not contain fulltext A boy with recurrent episodes of hypoglycaemia and ataxia, microcephaly, mental retardation, permanent lactic acidaemia, intermittent 2-oxoglutaric aciduria as well as elevation of serum branched chain amino acids was diagnosed with dihydrolipoamide dehydrogenase (E3) deficiency. Analysis of genomic DNA revealed compound heterozygosity for two novel mutations: I393T in exon 11, located at the interface domain of the protein and possibly interfering with its dimerisation, and IVS9+1G>A located at a consensus splice site. A heterozygous polymorphism was also detected. In the patient's cDNA the I393T mutation and the polymorphism appeared to be homozygous, indica…

MaleHeterozygoteMutation MissensePyruvate Dehydrogenase ComplexGene mutationBiologyCompound heterozygosityLoss of heterozygositymedicineHumansLeigh diseaseMuscle SkeletalDihydrolipoamide DehydrogenaseGeneticsSplice site mutationDihydrolipoamide dehydrogenasePyruvate Dehydrogenase (Lipoamide)Fibroblastsmedicine.diseasePyruvate dehydrogenase complexRenal disorders [UMCN 5.4]Genetic defects of metabolism [UMCN 5.1]Child PreschoolPediatrics Perinatology and Child HealthRNA Splice SitesLeigh DiseaseCellular energy metabolism [UMCN 5.3]
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Food Intake Adaptation to Dietary Fat Involves PSA-Dependent Rewiring of the Arcuate Melanocortin System in Mice

2012

International audience; Hormones such as leptin and ghrelin can rapidly rewire hypothalamic feeding circuits when injected into rodent brains. These experimental manipulations suggest that the hypothalamus might reorganize continually in adulthood to integrate the metabolic status of the whole body. In this study, we examined whether hypothalamic plasticity occurs in naive animals according to their nutritional conditions. For this purpose, we fed mice with a short-term high-fat diet (HFD) and assessed brain remodeling through its molecular and functional signature. We found that HFD for 3 d rewired the hypothalamic arcuate nucleus, increasing the anorexigenic tone due to activated pro-opio…

MaleMESH: Signal TransductionPro-Opiomelanocortin[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionSYNAPTIC INPUT ORGANIZATIONMESH: Energy IntakeWeight GainMESH: Mice KnockoutMice0302 clinical medicineMESH : Sialic AcidsNPY/AGRP NEURONSMESH: Pro-OpiomelanocortinMESH: AnimalsMESH : Neuronal PlasticityMESH: Neuronal PlasticityPLASTICITYMESH : Pro-OpiomelanocortinMESH : Adaptation PhysiologicalMice KnockoutFEEDING CIRCUITSMESH : Organ Culture TechniquesINSULIN-RESISTANCE0303 health sciencesNeuronal PlasticityPOLYSIALIC ACIDGeneral NeuroscienceLeptinMESH: Energy Metabolismdigestive oral and skin physiologyINDUCED OBESITYMESH : SialyltransferasesMESH : Weight GainArticlesAdaptation PhysiologicalMESH : Mice TransgenicBODY-WEIGHTMESH: Dietary FatsHypothalamusCELL-ADHESION MOLECULEMESH: Weight GainGhrelinENERGY-BALANCEMelanocortinhormones hormone substitutes and hormone antagonistsSignal Transductionmedicine.medical_specialtyMESH: Mice TransgenicMESH : MaleMESH: SialyltransferasesMESH: Arcuate NucleusMice TransgenicMESH : Mice Inbred C57BLBiologyMESH : Arcuate NucleusMESH: Sialic Acids03 medical and health sciencesOrgan Culture TechniquesInsulin resistanceMESH: Mice Inbred C57BLArcuate nucleusInternal medicineMESH : MicemedicineAnimalsMESH: Mice030304 developmental biologyMESH : Signal TransductionArcuate Nucleus of HypothalamusMESH : Energy Intakemedicine.diseaseDietary FatsMESH: Adaptation PhysiologicalSialyltransferasesMESH: Organ Culture TechniquesMESH: MaleMice Inbred C57BLMESH : Energy MetabolismEndocrinologyMESH: Nerve NetSialic AcidsMESH : Nerve NetMESH : Mice KnockoutMESH : AnimalsNerve NetEnergy IntakeEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Dietary Fats030217 neurology & neurosurgeryHomeostasisHormoneThe Journal of Neuroscience
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Effects of a high-fat diet on energy metabolism and ROS production in rat liver.

2011

International audience; BACKGROUND & AIMS: A high-fat diet affects liver metabolism, leading to steatosis, a complex disorder related to insulin resistance and mitochondrial alterations. Steatosis is still poorly understood since diverse effects have been reported, depending on the different experimental models used. METHODS: We hereby report the effects of an 8 week high-fat diet on liver energy metabolism in a rat model, investigated in both isolated mitochondria and hepatocytes. RESULTS: Liver mass was unchanged but lipid content and composition were markedly affected. State-3 mitochondrial oxidative phosphorylation was inhibited, contrasting with unaffected cytochrome content. Oxidative…

Mitochondrial ROSMaleTranscription GeneticMESH : Reactive Oxygen SpeciesMitochondria LiverMESH : HepatocytesMitochondrionOxidative PhosphorylationMESH: Hepatocytes0302 clinical medicineMESH: Membrane Potential MitochondrialCitrate synthaseMESH: AnimalsBeta oxidationMESH : Electron Transport2. Zero hungerMembrane Potential Mitochondrial0303 health sciencesMESH : RatsAdenine nucleotide translocatorMESH: Energy MetabolismMESH: Reactive Oxygen SpeciesLipidsBiochemistryLiverMESH: Dietary FatsMitochondrial matrix030220 oncology & carcinogenesisBody CompositionMESH : Oxidative PhosphorylationATP–ADP translocaseMESH: Mitochondria LiverMESH: RatsMESH : Body CompositionMESH : MaleOxidative phosphorylationBiologyMESH : Rats WistarElectron Transport03 medical and health sciencesMESH: Oxidative Phosphorylation[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRats WistarMESH: Electron Transport[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biology030304 developmental biologyHepatologyMESH: Transcription GeneticMESH : Transcription GeneticMESH : LiverMESH : LipidsMESH: Body CompositionMESH: Rats WistarMESH: LipidsDietary FatsMESH: MaleRatsMESH : Energy MetabolismMESH : Membrane Potential MitochondrialMESH : Mitochondria Liverbiology.proteinHepatocytesMESH : AnimalsEnergy MetabolismReactive Oxygen SpeciesMESH : Dietary FatsMESH: Liver
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